Clustering of Alpers disease mutations and catalytic defects in biochemical variants reveal new features of molecular mechanism of hte human mitochondrial replicase, Pol gamma

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Euro , L , Farnum , G A , Palin , E , Suomalainen , A & Kaguni , L S 2011 , ' Clustering of Alpers disease mutations and catalytic defects in biochemical variants reveal new features of molecular mechanism of hte human mitochondrial replicase, Pol gamma ' , Nucleic Acids Research , vol. 39 , no. 21 , pp. 9072-9084 . https://doi.org/10.1093/nar/gkr618

Titel: Clustering of Alpers disease mutations and catalytic defects in biochemical variants reveal new features of molecular mechanism of hte human mitochondrial replicase, Pol gamma
Författare: Euro, Liliya; Farnum, Gregory A.; Palin, Eino; Suomalainen, Anu; Kaguni, Laurie S.
Upphovmannens organisation: Research Programme for Molecular Neurology
Neurologian yksikkö
Datum: 2011
Språk: eng
Sidantal: 13
Tillhör serie: Nucleic Acids Research
ISSN: 0305-1048
DOI: https://doi.org/10.1093/nar/gkr618
Permanenta länken (URI): http://hdl.handle.net/10138/162362
Subject: DNA-POLYMERASE-GAMMA
PROGRESSIVE EXTERNAL OPHTHALMOPLEGIA
VIRION RNA-POLYMERASE
ACCESSORY SUBUNIT
STRUCTURAL BASIS
SACCHAROMYCES-CEREVISIAE
MULTIPLE DELETIONS
CRYSTAL-STRUCTURE
ESCHERICHIA-COLI
W748S MUTATION
3124 Neurology and psychiatry
Referentgranskad: Ja
Licens: cc_by_nc
Användningsbegränsning: openAccess
Parallelpublicerad version: publishedVersion


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